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Primary treatment of small vessel vasculitis damage

The health system is mixed with anti-human immunoglobulin in the MPO and PR3ANCA unique antibodies,[link widoczny dla zalogowanych], inhibition of AN-cA closed and its corresponding antigen in combination. In addition, IVIg can inhibit the AN-CA induced release of neutrophil activity and the ability of cytokines, with non-specific inhibition of macrophages and T cells release cytokines leaching, such as TNFa and IL-1 and so on, these mechanisms are possible and Ⅳ Ig with anti-inflammatory effects on vascular inflammation related. Ⅳ Ig more common adverse reactions such as headaches, back pain and fever. Transaminase elevation, etc., most of the light,[link widoczny dla zalogowanych], a small number of patients have elevated serum creatinine, but are reversible. 3.2 anti-lymphocyte antibody combined with anti-CD4 and CD52 humanized monoclonal antibody (humanisedmonoclonalantibod-ies) the treatment of refractory Wegener's granulomatosis some induced remission, the rapid onset of this therapy, and contribute to ACTH and the withdrawal of cytotoxic drugs, adverse reactions rarely infected. The main treatment mechanism and the elimination of circulating lymphocytes, the immune balance adjustments related. Try this treatment for the future treatment of primary small vessel vasculitis or autoimmune diseases,[link widoczny dla zalogowanych], provide a new specific way. 3.3 anti-infection treatment infection (including bacteria, viruses, etc.) is an important primary small vessel vasculitis patients with complications and cause of death, are often relapse triggers. Attention, identification and diagnosis of infection of primary small vessel vasculitis is an important part of treatment. Study confirmed that patients with Wegener's granulomatosis carry Staphylococcus aureus nasal (S.aureus) carriers carry less seven times its high recurrence rate, recurrence of Wegener's granulomatosis is an important reason. A controlled, randomized studies have shown that application of cotrimoxazole Clear S. aureus significantly reduce the recurrence of Wegener's granulomatosis. Application dose of sulfamethoxazole Hey azole (sulfamethoxa-zole) 800mg and trimethoprim (trimethroprim) 160mg, 2 times a day, 24 months. Recently reported that topical application of nasal mupirocin (Mupirocin) has a good clear S. aureu8 effect on kidney damage Wegener's granulomatosis and cotrimoxazole can cause adverse reactions in patients receiving treatment can be applied. 3.4 Application-specific ELISA specific ANCA target antigen (such as recombinant MPO) binds to the resin for adsorption of the corresponding serum ANCA. Small number of cases the experimental treatment has been reported in the completion of a variety of artificial re-ANCA target antigen-specific ELISA based on the specific method is a worthwhile way to explore the specificity of 3.5 dialysis treatment and kidney transplantation by about 10% of a 20% of primary small vessel vasculitis patients with irreversible end-stage renal failure to enter, need to rely on long-term dialysis treatment or kidney transplantation. Although patients with end-stage renal failure after renal transplantation, there is further involvement of the small number of graft reports,[link widoczny dla zalogowanych], but most scholars still believe that renal transplantation is a better way of renal replacement therapy, but should choose activities in the absence of disease, lesions of the . 4 prognosis of domestic and foreign research results were, in general, ANCA associated necrotizing crescentic glomerulonephritis prognosis than Goodpastme syndrome, RPGNI type and RPGNII type relatively good. About 20% to 40% of patients diagnosed or people's homes need to help sustain life,[link widoczny dla zalogowanych], dialysis, about 40% to 50% of patients have varying degrees of renal dysfunction. The immunosuppression treatment, about 60% to 80% of patients requiring dialysis can be out of dialysis, renal function was fully restored even really into irreversible end-stage renal failure patients is about 10% to 15%. Anti-neutrophil cytoplasmic antibody associated vasculitis etiology and pathogenesis of the Sino-Japanese Friendship Hospital, Beijing Guochun Division of Rheumatology (100029) anti-neutrophil cytoplasm antibody associated systemic vasculitis (ANCAassociatedsystemicvasculitis, from SV) major is Wegener's granulomatosis (WG), Churg-Strauss syndrome (CSS) and microscopic polyangiitis type (microscopicpolyangiitis, MPA) three diseases. Their clinical, pathological and pathogenesis have many similar characteristics, that is usually manifested ANCA positive serum; often pulmonary and renal involvement; tissue pathology of small blood vessels (small veins, capillaries and small arteries) involved based. Therefore, ChapelHill vasculitis classification criteria established by the Conference in which they were classified as vasculitis of small vessel involvement (Table 1). Buildings in recent years about the cause of AASV pathogenesis of a
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