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Interferon in the treatment of malignant diseases of the blood

apyofCMLwithinterferonsCancerSurvl989; 8 (4): 7934. MaglavePB, eta1. Effectofrecomrahinantgamma · I83 · interferononchronicmyelogenousleukemiabonemarrowprogenitorsExpHaematol1987; I5: 33l5McglavePB, eta1. AutologoustransplantationforCMLusingmarrowtreatmentexvivowithrecombinanthumaninterferongammaBoneMar-ToTraesNatttad. ott1990; 6:115.6. FDonKA. etal1. grad. BceI17cutaneousTcell}? A f anti-thyroid lympholytieleukemiaSeminOnco! I986: 13 (3si1ppl2): 357CooperMR. etalInterferonsinthetreatmentofmultiolemyelomaCancer1987; 59:5948 GaleRP, etalChroniclymphocyticleukemia: rcenladvaRc ~ sinbiologyandtreatment. AnnIn · Graves disease is the most common form of hyperthyroidism type,[link widoczny dla zalogowanych], a variety of clinical and experimental studies have shown that the disease is an autoimmune disease, immune cell function and mutual relationship between the ratio of change, but also for the existence of the composition of thyroid autoantibodies. Application of anti-thyroid drugs and I treatment, can cause changes in immune parameters. The following summarizes the two patients of treatment on Graves's own immune cells and antibody concentration. First, the impact of anti-thyroid drugs commonly used anti-thyroid drugs are divided into classes and imidazole sulfur legs, which can inhibit the peroxidase activation of inorganic iodine to prevent thyroid hormone synthesis, thus treatment of hyperthyroidism. In recent years, found the drugs on the immune system also has multiple effects. Graves patients with immune abnormalities exists, briefly speaking with: T cells reduced the total number and Ts, Ts dysfunction; B cell activation, resulting in a variety of autoantibodies against thyroid tissue, which affects thyroid gland function. l_ effect on the immune cells of T cells in this study more. Gr ~ ves the patient's peripheral blood lymphocytes (PBL),[link widoczny dla zalogowanych], before treatment, CD cells were significantly decreased activation of T cells (TA) significantly increased levels after treatment of hyperthyroidism, the former increased. The latter decline, the Ministry to normal. Li Qiongfang determined Grave8 other T cell subsets of patients before and after treatment change. Before treatment,[link widoczny dla zalogowanych], CD4 cells and CDR cells were significantly decreased, CD8 cells decreased particularly, CD and CD ratio has increased significantly; by anti-thyroid drug treatment after recovery, CD and cD cells were significantly increased, the ratio be reduced accordingly. In vitro cell culture studies have proved this trend, Wilson and other patients through the PBL cultured Graves, we found that T cells and Ts cells decreased significantly,[link widoczny dla zalogowanych], but with methimazole or propylthiouracil were cultured with increased. But there are of the view that, Graves Ts cells in peripheral blood, TH compared with normal cells and their ratio, no significant difference, but also in anti-thyroid drugs and no significant change during treatment. . . 2. Many changes in the concentration of autoantibodies found in Graves patients there is a higher concentration of the body's own antibodies, such as TSH receptor antibody, thyroid stimulating antibody (TSAb), thyroid binding inhibitory immunoglobulin (TBII), thyroid microsomal antibody , thyroglobulin antibody DNA antibodies, T, T antibodies to study more patients with Graves hyperthyroidism the TSH receptor antibodies. These antibodies are anti-thyroid drug treatment, have different levels of reduction. In addition, Wilson,[link widoczny dla zalogowanych], etc. are also carried out in vitro studies in this area, they used the Graves patients with PBL in vitro with methimazole or propylthiouracil with cultured with PWM stimulation, found that the culture medium
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